Corticospinal tract hyperintensity

Evidence-based neurology checklist on corticospinal tract hyperintensity: Metabolic causes Krabbe disease X-linked adrenoleukodystrophy Cerebrotendinous xanthomatosis (CTX) Hepatic encephalopathy Neuromuscular causes Infective causes Autoimmune causes Other causes

Metabolic causes

  • Krabbe disease
  • X-linked adrenoleukodystrophy
  • Cerebrotendinous xanthomatosis (CTX)
  • Hepatic encephalopathy

Neuromuscular causes

Infective causes

Autoimmune causes

Other causes

References

  1. Pruvost-Robieux E, Yeung J, Sudacevschi V, Cordoliani Y, De Malherbe M, Pico F. Reversible corticospinal tract hyperintensities in neurologic Lyme disease. Neurology 2016; 87:548-549. 
  2. Sehgal R, Sharma S, Sankhyan N, Kumar A, Gulati S. Selective corticospinal tract involvement in late-onset Krabbe disease. Neurology 2011; 77:e20. 
  3. Bang H, Lee HY, Kim BR, Lee IS, Jung H, Koh SE, Lee J. Hepatic encephalopathy with corticospinal tract involvement demonstrated by diffusion tensor tractography. Ann Rehabil Med 2015; 39:138-141.
  4. Hecht MJ, Fellner F, Fellner C, Hilz MJ, Heuss D, Neundörfer B. MRI-FLAIR images of the head show corticospinal tract alterations in ALS patients more frequently than T2-, T1- and proton-density-weighted images. J Neurol Sci 2001; 186:37-44.
  5. Jin J, Hu F, Zhang Q, Jia R, Dang J. Hyperintensity of the corticospinal tract on FLAIR: A simple and sensitive objective upper motor neuron degeneration marker in clinically verified amyotrophic lateral sclerosis. J Neurol Sci 2016; 367:177-183.
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